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Advanced search Search history. Browse titles authors subjects uniform titles series callnumbers dewey numbers starting from optional. See what's been added to the collection in the current 1 2 3 4 5 6 weeks months years. Your reader barcode: Your last name:. Cite this Email this Add to favourites Print this page. So the possible harmful effects in morpho-functional integrity of this organ may after the hemo-liquor barrier and indirectly the blood brain barrier and induce to pre and postnatal disorders in Central Nervous System CRACIUN et al.

The changes observed as well as having observed in the animals of the group Ep of the our studies, are presumably due to a disturbance in hidroelectrolite homeostasis and the changes caused by alcohol in cerebral membranes are apropriate and complementary to this supposed hypothesis maybe being responsible, at least, for some of the pathologies in Central Nervous System in alcoholics CHECIU et al.

Thus, Group Ab animals that started to receive tap water again as the liquid diet after days of treatment showed a marked recovery of the characteristics of the choroid tissue as a whole. On the basis of the morphologic characteristics of this structure, we may assume that at least a partial recovery of its function also occurred. The partial regression of the ultrastructural changes observed in the choroid epithelium of the abstemious animals after days of treatment, takes us to suggest that has happened due to the abstinence of those animals to the alcohol, and that the regression was not only larger, possibly for the small space of time that gave him between the withdrawal of the alcohol and the sacrifice and analysis of the organ 60 days.

Anat, 16 2 , Alcoholismo; 2. Plexo coroideo; 4. Luis Fernando Tirapelli Depto. Ultrastructural changes in the mouse fetal neocortex following chronic maternal alcoholization. Recognizing alcoholism and its effects: a mine-guide.

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Basel, S. Karger AG, Ultrastructural changes in the mouse fetal choroid plexuses following chronic maternal alcoholization. The blood-brain barrier. The structure and function of nervous tissue. New York, Academic Press, Savier, A possible mechanism of action ethyl alcohol on the central nervous system. Fenniae, , The effect of acute alcoholism intoxication on the bood-brain barrier.

Prenatal exposure to ethanol alters lateral plasma membranes and gap junctions of newborn rat hepatocytes as revealed by freeze-fracture. Chronic ethanol consumption affects Filipin-Cholesterol complexes and intramembranous particles od synaptossomes of rat brain cortex. Alcoholism Clin. Potentation of the narcotic action and acute toxicity of alcohol by primary aromatic monoamines. Ultrastructural changes of choroid plexuses of lateral ventricles of rats Rattus norvegicus submitted to chronic experimental alcoholism.

Botucatu, Effects of acetylcholine analogues and ethanol on the respiration of brain cortex tissue in vitro. Alcohol 44, — Aoto, K. Fetal ethanol exposure activates protein kinase A and impairs shh expression in prechordal plate mesoderm cells in the pathogenesis of holoprosencephaly. Birth Defects Res. A Clin. Blader, P. Ethanol impairs migration of the prechordal plate in the zebrafish embryo. Boehm, S. Alcohol 14, — Bolnick, J. Apoptosis of alcohol-exposed human placental cytotrophoblast cells is downstream of intracellular calcium signaling.

Brault, V. Development , — Bunout, D. Nutritional and metabolic effects of alcoholism: their relationship with alcoholic liver disease. Nutrition 15, — CrossRef Full Text. Bupp Becker, S. Teratogenicity of ethanol in different chicken strains. Alcohol Alcohol. Cartwright, M. Stage dependent effects of ethanol on cranial neural crest cell development: partial basis for the phenotypic variations observed in fetal alcohol syndrome.

Ethanol-induced neural crest apoptosis is coincident with their endogenous death but is mechanistically distinct.

Carvan, M. III, Loucks, E. Ethanol effects on the developing zebrafish: neurobehavior and skeletal morphogenesis. Cavieres, M. Genetic and developmental modulation of cardiac deficits in prenatal alcohol exposure. Chen, S. Differential sensitivity of mouse neural crest cells to ethanol-induced toxicity.

Alcohol 20, 75— Chen, X. Over-expression of Nrf2 diminishes ethanol-induced oxidative stress and apoptosis in neural crest cells by inducing an antioxidant response. Debelak, K. Avian genetic background modulates the neural crest apoptosis induced by ethanol exposure. Debelak-Kragtorp, K. Ethanol-induced cephalic apoptosis requires phospholipase C-dependent intracellular calcium signaling.

Downing, C. Ethanol teratogenesis in five inbred strains of mice. Quantitative trait locus mapping for ethanol teratogenesis in BXD recombinant inbred mice. Dubourg, C. Rare Dis. Dunty, W. Selective vulnerability of embryonic cell populations to ethanol-induced apoptosis: implications for alcohol-related birth defects and neurodevelopmental disorder.


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Pubmed Abstract Pubmed Full Text. Edenberg, H. Genetics and alcoholism. Ellies, D. Apoptosis of premigratory neural crest cells in rhombomeres 3 and 5: consequences for patterning of the branchial region. Flentke, G. III, and Smith, S. An evolutionarily-conserved mechanism of calcium-dependent neurotoxicity in a zebrafish model of FASD.

Fumagalli, S. The role of p53 in ribosomopathies. Garic, A. High-throughput transcriptome sequencing identifies candidate genetic modifiers of vulnerability to fetal alcohol spectrum disorders. Garic-Stankovic, A. Gilliam, D.

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Dose-related growth deficits in LS but not SS mice prenatally exposed to alcohol. Alcohol 13, 47— Goodlett, C. Genetic influences on brain growth restriction induced by developmental exposure to alcohol. Neurotoxicity 10, — Granneman, S. Ribosome biogenesis: of knobs and RNA processing. Cell Res. Green, M. Reprogramming of genetic networks during initiation of the fetal alcohol syndrome. Gutala, R. Microarray analysis of ethanol-treated cortical neurons reveals disruption of genes related to the ubiquitin-proteasome pathway and protein synthesis. Hassler, J. Effects of ethanol on the cytoskeleton of migrating and differentiating neural crest cells: possible role in teratogenesis.

Hong, M. Codon mutation and fetal ethanol exposure synergize to produce midline signaling defects and holoprosencephaly spectrum disorders in mice. PLoS Genet. Howard, R. Alcohol-binding sites in distinct brain proteins: the quest for atomic level resolution. Kietzman, H. K, and Lipinski, R.

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The teratogenic effects of prenatal ethanol exposure are exacerbated by Sonic Hedgehog or GLI2 haploinsufficiency in the mouse. Kilburn, B. Klingenberg, C. Prenatal alcohol exposure alters the patterns of facial asymmetry. Kohn, A. Cell Calcium 38, — Kouzoukas, D. Intracellular calcium plays a critical role in the alcohol-mediated death of cerebellar granule neurons. Kruse, J.

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Modes of p53 regulation. Cell , — Li, Y. Fetal alcohol exposure impairs Hedgehog cholesterol modification and signaling. Lipinski, R. Ethanol-induced face-brain dysmorphology patterns are correlative and exposure-stage dependent. Loucks, E. Strain-dependent effects of developmental ethanol exposure in zebrafish.

MacDonald, B. Cell 17, 9— McCarthy, N. Pdgfra protects against ethanol-induced craniofacial defects in a zebrafish model of FASD. Narla, A. Ribosomopathies: human disorders of ribosome dysfunction. Blood , — Oyedele, O. Nuanced but significant: how ethanol perturbs avian cranial neural crest cell actin cytoskeleton, migration and proliferation. Alcohol 47, — Rahman, S. Identification of novel ethanol-sensitive genes by expression profiling. Reimers, M.